January 28, 2015
The Anunnaki did not want to repeat the past mistakes that were made on Mars with regards to creating a labor force. Success would be the creation of an inferior copy of themselves for the mining of gold. They did not want a labor force that was an intellectual equal. To achieve their goals for a controllable workforce they deactivated L-gulonolactone oxidase molecule the fourth and last pathway in the biochemical synthesis of ascorbic acid from glucose. One of the effects of shutting down this enzyme was the acceleration of neural development. The Anunnaki altered the genotype by activating a “sheeple gene” which retarded any neural enhancements. Having a high IQ and mental telepathy were enhancements that were considered undesirable.
Gamma-rays are the most energetic form of light and are produced by the hottest regions of the universe. Things like supernova explosions (the way massive stars die), neutron stars and pulsars, and black holes are all sources of cosmic gamma-rays. Linus Pauling PhD and Irwin Stone PhD were of the belief that stellar rays such as gamma rays could be responsible for inactivating the L-gulonolactone oxidase molecule. It is easy to imagine celestial gamma rays bathing the entire Earth. What is perplexing is that anthropoid primates, guinea pigs, Fruit eating bat (Pteropus Medius), Passeriform (perching) birds and Teleost (ray-finned) fishes do not produce their own ascorbic acid. Why isn’t this list a lot bigger? It is within the realm of possibilities that gamma rays could have deactivated the L-gulonolactone oxidase molecule in many more plants and animals that science is not aware of. Dr. Chatterjee has stated that there have been cases where some species have lost the ability to synthesize ascorbic acid only to regain it again.
A hypothesis will be put forth that guinea pigs, Fruit eating bat (Pteropus Medius), Passeriform (perching) birds and Teleost (ray-finned) were a primary food source of those genetically modified hominoids (The fact that Teleost fishes could have been a primary food source for hominoids may give credence to Elaine Morgan’s “Aquatic Ape Theory”). When L-gulonolactone oxidase was turned off this made the species susceptible to a whole range of sub-clinical scurvy type diseases. The only benefit from this deactivation was accelerated neural growth. The discovery of a “sheeple gene” in the genome was important. They wanted the Homo sapiens to follow orders but not be too smart as to pose a threat to the Anunnaki aristo-elites. A measurement of cranial capacity is an indicator of their mental capabilities. They needed a field stress test to see what this first generation of Homo sapiens was capable of.
Robert F. Cathcart M.D. made an assertion that during periods of great evolutionary stress animals without L-gulonolactone oxidase enzyme could excel in their environment to the extent that those animals with the gene became extinct. The deactivation of the L-gulonolactone oxidase molecule in the guinea pigs, Fruit eating bat, Passeriformes birds and Teleost fishes was an environmental stress test for adaptability. Those that could find an alternative source for ascorbic acid would continue to breed while those that could not would face extinction.
They created a docile workforce that was just smart enough to take orders and execute them. A deactivated GLO enzyme ensured that the workforce had a short lifespan. A shortened lifespan guaranteed a regular turnover of the workforce. This inhibited any kind of unionizing of the labor force. The deactivated enzyme made this labor force susceptible to a whole range of diseases from scurvy to subclinical scurvy type diseases from the common cold, heart disease and cancer. Susceptibility to diseases was increased by the fact that the genetically designed labor force could not synthesize their own ascorbic acid in addition to vitamins A, B1 (thiamine), B2 (riboflavin), B5 (pantothenic acid), B6 (pyridoxine), B7 (biotin), B9 (folate), B12 (cobalamin), E and K but are able to synthesize some vitamin B3 (niacin) not very efficiently and D.
When the Anunnaki no longer had use for their human slaves they thought that the deactivation of the L-gulonolactone enzyme along with environmental ascorbic acid’s property of being “somewhat unstable and transient” would kill off Homo sapiens 1.0 (Similar to what happened in the movie Jurassic Park where the lysine contingency was intended as a foolproof fallback plan to ensure that the dinosaurs never left the island: the dinosaurs were specially engineered to lack supplemental enzymes in their diet, and were incapable of producing the amino acid lysine. This meant that, unless they were artificially provided with lysine through their regular diet, they would slip into a comatose state and die) and hominoid evolution would eventually resume its normal evolutionary course.
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